Comparative evidence
Compare conditions
Put two canonical disease records side by side. The result is a shareable, deterministic comparison of what is shared, what is unique, and how much evidence each record currently carries.
- 19%
- weighted overlap
- 15
- shared signals
Comparison
Graves' DiseaseandType 2 Diabetes
0 references occur in both reconciled evidence sets. Shared signals do not establish shared causes, diagnoses, or treatments.
- 22
- Graves' Disease references
- 30
- Type 2 Diabetes references
- 0
- shared references
Metals depleted or redistributed
1 sharedTaxa enriched
4 sharedShared
Only in Graves' Disease
Only in Type 2 Diabetes
Taxa depleted
5 sharedShared
Only in Graves' Disease
Only in Type 2 Diabetes
Host response elevated
1 sharedShared
Only in Graves' Disease
Only in Type 2 Diabetes
LipopolysaccharideInflammatory CytokinesTNF-alpha (Tumor Necrosis Factor Alpha)IL 6FerritinCeruloplasmin
Host response depleted
0 sharedEcological state
0 sharedShared
None indexed
Only in Graves' Disease
Intestinal PermeabilityTh17 Treg ImbalanceMolecular MimicryEstrogen RecirculationSCFA DeficiencyLPS TranslocationBidirectional Gut Thyroid Axis
Only in Type 2 Diabetes
DysbiosisSCFA DepletionLPS ElevationBile Acid DysmetabolismImpaired Intestinal BarrierLipid PeroxidationReduced Microbial DiversityEndotoxemiaGut Barrier DisruptionBile Acid DysregulationIron Overload Oxidative StressFungal Overgrowth
Virulence and functional enzymes
1 sharedShared
Only in Graves' Disease
Only in Type 2 Diabetes
LipopolysaccharideCholine TMA LyaseBile Acid DehydrataseCarbohydrate Fermentation EnzymesLPS Biosynthesis EnzymesNickel-UreaseTMA Lyase CutC CutD