Comparative evidence
Compare conditions
Put two canonical disease records side by side. The result is a shareable, deterministic comparison of what is shared, what is unique, and how much evidence each record currently carries.
- 11%
- weighted overlap
- 9
- shared signals
Comparison
Type 2 DiabetesandPremenstrual Dysphoric Disorder
0 references occur in both reconciled evidence sets. Shared signals do not establish shared causes, diagnoses, or treatments.
- 30
- Type 2 Diabetes references
- 6
- Premenstrual Dysphoric Disorder references
- 0
- shared references
Metals depleted or redistributed
2 sharedShared
Only in Type 2 Diabetes
Only in Premenstrual Dysphoric Disorder
None indexed
Taxa enriched
1 sharedShared
Only in Type 2 Diabetes
Only in Premenstrual Dysphoric Disorder
Anaerotaenia
Taxa depleted
2 sharedShared
Only in Type 2 Diabetes
Only in Premenstrual Dysphoric Disorder
Host response elevated
1 sharedShared
Only in Type 2 Diabetes
Only in Premenstrual Dysphoric Disorder
Host response depleted
2 sharedShared
Only in Type 2 Diabetes
Only in Premenstrual Dysphoric Disorder
Ecological state
1 sharedShared
SCFA Depletion
Only in Type 2 Diabetes
DysbiosisLPS ElevationBile Acid DysmetabolismImpaired Intestinal BarrierLipid PeroxidationReduced Microbial DiversityEndotoxemiaGut Barrier DisruptionBile Acid DysregulationIron Overload Oxidative StressFungal Overgrowth
Only in Premenstrual Dysphoric Disorder
Tryptophan Kynurenine ShuntingEstrobolome DysfunctionGABA A Receptor ModulationLuteal Phase InflammationBeta Glucuronidase DysregulationEstrogen RecirculationBeta Glucuronidase ActivityCircadian Rhythm DisruptionMelatonin Amplitude Reduction
Virulence and functional enzymes
0 sharedShared
None indexed
Only in Type 2 Diabetes
LipopolysaccharideCholine TMA LyaseBile Acid DehydrataseCarbohydrate Fermentation EnzymesSiderophoresLPS Biosynthesis EnzymesNickel-UreaseTMA Lyase CutC CutD