Comparative evidence
Compare conditions
Put two canonical disease records side by side. The result is a shareable, deterministic comparison of what is shared, what is unique, and how much evidence each record currently carries.
- 12%
- weighted overlap
- 8
- shared signals
Comparison
Type 2 DiabetesandCeliac Disease
0 references occur in both reconciled evidence sets. Shared signals do not establish shared causes, diagnoses, or treatments.
- 30
- Type 2 Diabetes references
- 8
- Celiac Disease references
- 0
- shared references
Metals depleted or redistributed
2 sharedTaxa enriched
1 sharedShared
Only in Type 2 Diabetes
Only in Celiac Disease
Taxa depleted
3 sharedShared
Only in Type 2 Diabetes
Only in Celiac Disease
Host response elevated
0 sharedShared
None indexed
Only in Type 2 Diabetes
LipopolysaccharideHepcidinInflammatory CytokinesTNF-alpha (Tumor Necrosis Factor Alpha)IL 6FerritinCeruloplasmin
Only in Celiac Disease
Host response depleted
1 sharedEcological state
0 sharedShared
None indexed
Only in Type 2 Diabetes
DysbiosisSCFA DepletionLPS ElevationBile Acid DysmetabolismImpaired Intestinal BarrierLipid PeroxidationReduced Microbial DiversityEndotoxemiaGut Barrier DisruptionBile Acid DysregulationIron Overload Oxidative StressFungal Overgrowth
Only in Celiac Disease
Villous AtrophyIncreased Intestinal PermeabilityGluten Mediated Tight Junction DisassemblyNickel Allergic Contact Mucositis
Virulence and functional enzymes
0 sharedShared
None indexed
Only in Type 2 Diabetes
LipopolysaccharideCholine TMA LyaseBile Acid DehydrataseCarbohydrate Fermentation EnzymesSiderophoresLPS Biosynthesis EnzymesNickel-UreaseTMA Lyase CutC CutD