Comparative evidence
Compare conditions
Put two canonical disease records side by side. The result is a shareable, deterministic comparison of what is shared, what is unique, and how much evidence each record currently carries.
- 13%
- weighted overlap
- 10
- shared signals
Comparison
Type 1 DiabetesandUlcerative Colitis
0 references occur in both reconciled evidence sets. Shared signals do not establish shared causes, diagnoses, or treatments.
- 20
- Type 1 Diabetes references
- 6
- Ulcerative Colitis references
- 0
- shared references
Metals depleted or redistributed
2 sharedTaxa enriched
2 sharedShared
Only in Type 1 Diabetes
Only in Ulcerative Colitis
Taxa depleted
3 sharedShared
Only in Type 1 Diabetes
BifidobacteriumSubdoligranulumEubacterium rectalePrevotellaLactobacillusClostridium Clusters IV/XIVaBlautiaEubacterium eligensClostridium
Only in Ulcerative Colitis
Host response elevated
1 sharedShared
Only in Type 1 Diabetes
ZnT8 AutoantibodiesGADAIA 2AInsulin AutoantibodiesType 1 IFN SignatureTLR4 NF KB ActivationFerritinAnti ZnT8 Autoantibodies
Only in Ulcerative Colitis
Host response depleted
1 sharedEcological state
0 sharedShared
None indexed
Only in Type 1 Diabetes
Butyrate SCFA Ratio ShiftLeaky Gut Precedes OnsetBacterial Translocation To PLNMucin Layer ThinningImmunoinhibitory LPSCVB4 Microbiome SynergyTwo Step Temporal ModelMicrobiome Changes Precede AutoantibodiesBacteroidetes Firmicutes Ratio InversionGut Barrier CompromiseLPS Translocation To PLNSCFA GPR43 Axis DisruptionTreg DepletionViral Dysbiosis TriggerDevelopmental Vulnerability Window
Only in Ulcerative Colitis
Iron Pathobiont Feedback LoopButyrate DepletionMucosal UlcerationBarrier DysfunctionTh17 Treg ImbalanceMucin Degradation
Virulence and functional enzymes
0 sharedShared
None indexed
Only in Type 1 Diabetes
LPS Biosynthesis GenesIron Acquisition FecB IsdDEFType VI Secretion SystemsDNA Adenine MethyltransferasesLPS BiosynthesisBeta-Glucuronidase