Comparative evidence
Compare conditions
Put two canonical disease records side by side. The result is a shareable, deterministic comparison of what is shared, what is unique, and how much evidence each record currently carries.
- 12%
- weighted overlap
- 8
- shared signals
Comparison
Celiac DiseaseandType 2 Diabetes
0 references occur in both reconciled evidence sets. Shared signals do not establish shared causes, diagnoses, or treatments.
- 8
- Celiac Disease references
- 30
- Type 2 Diabetes references
- 0
- shared references
Metals depleted or redistributed
2 sharedTaxa enriched
1 sharedShared
Only in Celiac Disease
Only in Type 2 Diabetes
Taxa depleted
3 sharedShared
Only in Celiac Disease
Only in Type 2 Diabetes
Host response elevated
0 sharedShared
None indexed
Only in Celiac Disease
Only in Type 2 Diabetes
LipopolysaccharideHepcidinInflammatory CytokinesTNF-alpha (Tumor Necrosis Factor Alpha)IL 6FerritinCeruloplasmin
Host response depleted
1 sharedEcological state
0 sharedShared
None indexed
Only in Celiac Disease
Villous AtrophyIncreased Intestinal PermeabilityGluten Mediated Tight Junction DisassemblyNickel Allergic Contact Mucositis
Only in Type 2 Diabetes
DysbiosisSCFA DepletionLPS ElevationBile Acid DysmetabolismImpaired Intestinal BarrierLipid PeroxidationReduced Microbial DiversityEndotoxemiaGut Barrier DisruptionBile Acid DysregulationIron Overload Oxidative StressFungal Overgrowth
Virulence and functional enzymes
0 sharedShared
None indexed
Only in Celiac Disease
Only in Type 2 Diabetes
LipopolysaccharideCholine TMA LyaseBile Acid DehydrataseCarbohydrate Fermentation EnzymesSiderophoresLPS Biosynthesis EnzymesNickel-UreaseTMA Lyase CutC CutD