Comparative evidence
Compare conditions
Put two canonical disease records side by side. The result is a shareable, deterministic comparison of what is shared, what is unique, and how much evidence each record currently carries.
- 20%
- weighted overlap
- 14
- shared signals
Comparison
Type 2 DiabetesandRheumatoid Arthritis
0 references occur in both reconciled evidence sets. Shared signals do not establish shared causes, diagnoses, or treatments.
- 30
- Type 2 Diabetes references
- 7
- Rheumatoid Arthritis references
- 0
- shared references
Metals depleted or redistributed
0 sharedTaxa enriched
2 sharedShared
Only in Type 2 Diabetes
Only in Rheumatoid Arthritis
Taxa depleted
4 sharedShared
Only in Type 2 Diabetes
Only in Rheumatoid Arthritis
None indexed
Host response elevated
2 sharedShared
Only in Type 2 Diabetes
Only in Rheumatoid Arthritis
Host response depleted
1 sharedEcological state
0 sharedShared
None indexed
Only in Type 2 Diabetes
DysbiosisSCFA DepletionLPS ElevationBile Acid DysmetabolismImpaired Intestinal BarrierLipid PeroxidationReduced Microbial DiversityEndotoxemiaGut Barrier DisruptionBile Acid DysregulationIron Overload Oxidative StressFungal Overgrowth
Only in Rheumatoid Arthritis
Mucosal Origin AutoimmunityBarrier DysfunctionMolecular MimicryTh17 DominanceMetal VitD Bone AxisSoil Food Chain Exposure
Virulence and functional enzymes
2 sharedShared
SiderophoresLPS Biosynthesis Enzymes
Only in Type 2 Diabetes
LipopolysaccharideCholine TMA LyaseBile Acid DehydrataseCarbohydrate Fermentation EnzymesNickel-UreaseTMA Lyase CutC CutD
Only in Rheumatoid Arthritis
Metalloproteinases