Comparative evidence
Compare conditions
Put two canonical disease records side by side. The result is a shareable, deterministic comparison of what is shared, what is unique, and how much evidence each record currently carries.
- 20%
- weighted overlap
- 14
- shared signals
Comparison
Rheumatoid ArthritisandType 2 Diabetes
0 references occur in both reconciled evidence sets. Shared signals do not establish shared causes, diagnoses, or treatments.
- 7
- Rheumatoid Arthritis references
- 30
- Type 2 Diabetes references
- 0
- shared references
Metals depleted or redistributed
0 sharedTaxa enriched
2 sharedShared
Only in Rheumatoid Arthritis
Only in Type 2 Diabetes
Taxa depleted
4 sharedShared
Only in Rheumatoid Arthritis
None indexed
Only in Type 2 Diabetes
Host response elevated
2 sharedShared
Only in Rheumatoid Arthritis
Only in Type 2 Diabetes
Host response depleted
1 sharedEcological state
0 sharedShared
None indexed
Only in Rheumatoid Arthritis
Mucosal Origin AutoimmunityBarrier DysfunctionMolecular MimicryTh17 DominanceMetal VitD Bone AxisSoil Food Chain Exposure
Only in Type 2 Diabetes
DysbiosisSCFA DepletionLPS ElevationBile Acid DysmetabolismImpaired Intestinal BarrierLipid PeroxidationReduced Microbial DiversityEndotoxemiaGut Barrier DisruptionBile Acid DysregulationIron Overload Oxidative StressFungal Overgrowth
Virulence and functional enzymes
2 sharedShared
SiderophoresLPS Biosynthesis Enzymes
Only in Rheumatoid Arthritis
Metalloproteinases
Only in Type 2 Diabetes
LipopolysaccharideCholine TMA LyaseBile Acid DehydrataseCarbohydrate Fermentation EnzymesNickel-UreaseTMA Lyase CutC CutD