Comparative evidence

Compare conditions

Put two canonical disease records side by side. The result is a shareable, deterministic comparison of what is shared, what is unique, and how much evidence each record currently carries.

9%: weighted overlap
4: shared signals

First condition

Second condition

Comparison

EpilepsyandCeliac Disease

0 references occur in both reconciled evidence sets. Shared signals do not establish shared causes, diagnoses, or treatments.

9: Epilepsy references
8: Celiac Disease references
0: shared references

Metals elevated or accumulated

0 shared

Shared

None indexed

Only in Epilepsy

Copper Arsenic

Only in Celiac Disease

Nickel

Metals depleted or redistributed

3 shared

Shared

Iron Zinc Magnesium

Only in Epilepsy

None indexed

Only in Celiac Disease

Selenium Copper Calcium

Taxa enriched

0 shared

Shared

None indexed

Only in Epilepsy

Akkermansia muciniphila Streptococcus Enterococcus Prevotella Veillonella

Only in Celiac Disease

Escherichia coli Bacteroides fragilis Proteobacteria (Pseudomonadota)Staphylococcus aureus

Taxa depleted

1 shared

Host response elevated

0 shared

Shared

None indexed

Only in Epilepsy

Calprotectin (S100A8/A9)IL-1beta (Interleukin-1 Beta)IL 6TNF-alpha (Tumor Necrosis Factor Alpha)

Only in Celiac Disease

Zonulin

Host response depleted

0 shared

Shared

None indexed

Only in Epilepsy

Butyrate GABA (Gamma-Aminobutyric Acid)

Only in Celiac Disease

Glutathione (GSH)

Ecological state

0 shared

Shared

None indexed

Only in Epilepsy

SCFA DepletionOral Gut TranslocationMucin DegradationKynurenine Pathway ShuntingNeuroinflammation

Only in Celiac Disease

Villous AtrophyIncreased Intestinal PermeabilityGluten Mediated Tight Junction DisassemblyNickel Allergic Contact Mucositis

Virulence and functional enzymes

0 shared

Shared

None indexed

Only in Epilepsy

Beta GlucosidaseTryptophanaseIndoleamine 2 3 Dioxygenase Upregulation

Only in Celiac Disease

Bacterial ProteasesBeta-GlucuronidaseTissue Transglutaminase 2 Mimicry